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strain cvs-11  (ANSES laboratories)

 
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    ANSES laboratories strain cvs-11
    Strain Cvs 11, supplied by ANSES laboratories, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/strain+cvs-11/fixed+rabv+challenge+virus+standard+strain+cvs+27+anses+13+12/10__31489_slash_2025bmg1_slash_36___43-60-0-2
    Average 90 stars, based on 1 article reviews
    strain cvs-11 - by Bioz Stars, 2026-09
    90/100 stars

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    Article Title: Modes for chromatographic purification of immunoglobulin G (IgG) from the serum of horses hyperimmunized with rabies antigen
    Article Snippet: Immunoglobulin G (IgG) plays a key role in the body’s immune response, its high specificity to antigens and effectiveness in neutralizing pathogens make it a valuable tool in medical and scientific research.. Horses are a source of significant amounts of IgG, but their purification to a level suitable for clinical or scientific use requires specialized methods.. Development of chromatographic purification modes for immunoglobulin G (IgG) from the serum of horses hyperimmunized with rabies antigen will allow standardizing the technology in accordance with international quality and safety standards for the production and control of immunobiological medicines, vaccines and diagnostic test systems.



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    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and <t>RABV</t> (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.
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    Image Search Results


    The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and RABV (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.

    Journal: mSphere

    Article Title: LINC2781 enhances antiviral immunity against coxsackievirus B5 infection by activating the JAK-STAT pathway and blocking G3BP2-mediated STAT1 degradation

    doi: 10.1128/msphere.00062-25

    Figure Lengend Snippet: The characterization of LINC2781. (A) A schematic representation of the human LINC2781 transcribed from the XLOC_262866 gene; (B) SH-SY5Y and THP-1 cells were infected with CVB5 at increasing MOIs for 24 h or CVB5 (MOI = 1) for the indicated time intervals. The expression of LINC2781 was measured by RT-qPCR; (C) SH-SY5Y cells were transfected with increasing amounts of CVB5 RNA or plasmids (2 µg) encoding viral nonstructural proteins. The expression of LINC2781 was measured by RT-qPCR; (D) SH-SY5Y cells were infected with CVB5, CVA16, EV71, HSV-1, and RABV (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (E) SH-SY5Y cells were stimulated with different amounts of poly (I:C) or poly (dG:dC) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (F) Various cell lines were infected with CVB5 (MOI = 1) for 24 h. The expression of LINC2781 was measured by RT-qPCR; (G) Three-day-old BALB/c mice were infected with CVB5 (20 LD50) for 10 days. The expression of LINC2781 was measured by RT-qPCR. (H) SH-SY5Y cells were treated with increasing amounts of IFN-β, IFN-γ, LPS, and TNF-α for 24 h. The expression of LINC2781 was measured by RT-qPCR. Biologically independent experiments ( n = 3) were conducted, and all data were shown as mean ± SD. Student’s t -test was used to detect significant differences, with P ≤ 0.05 (*), P ≤ 0.01 (**), P ≤ 0.001 (***), and ns for no significant difference.

    Article Snippet: Coxsackievirus A16 (CVA16) strain 74/YN/2016 (GenBank: KY440934.1 ), enterovirus A71 (EV71) strain RA330/YN/CHN/2009 (GenBank: MK028135.1 ), human herpes virus 1 (HSV-1) isolate ZW6 (GenBank: KX424525.1 KX424525.1 ), and rabies virus CVS-11 (RABV, ATCC VR 959) were stored in our laboratory.

    Techniques: Infection, Expressing, Quantitative RT-PCR, Transfection